Epigenetic alteration of Wnt pathway antagonists in progressive glandular neoplasia of the lung

Julien D. F. Licchesi; William H. Westra; Craig M. Hooker; Emi O. Machida; Stephen B. Baylin; James G. Herman

doi:10.1093/carcin/bgn017

Epigenetic alteration of Wnt pathway antagonists in progressive glandular neoplasia of the lung

Julien D. F. Licchesi, William H. Westra, Craig M. Hooker, Emi O. Machida, Stephen B. Baylin, James G. Herman

Department of Life Sciences

Research output: Contribution to journal › Article › peer-review

119 Citations (SciVal)

Abstract

Atypical adenomatous hyperplasia (AAH) is now recognized as a precursor lesion from which lung adenocarcinomas arise and thus represents an ideal target for studying the early genetic and epigenetic alterations associated with lung tumorigenesis such as alterations of the Wnt pathway.

Original language	English
Pages (from-to)	895-904
Number of pages	10
Journal	Carcinogenesis
Volume	29
Issue number	5
Early online date	28 Feb 2008
DOIs	https://doi.org/10.1093/carcin/bgn017
Publication status	Published - May 2008

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title = "Epigenetic alteration of Wnt pathway antagonists in progressive glandular neoplasia of the lung",

abstract = "Atypical adenomatous hyperplasia (AAH) is now recognized as a precursor lesion from which lung adenocarcinomas arise and thus represents an ideal target for studying the early genetic and epigenetic alterations associated with lung tumorigenesis such as alterations of the Wnt pathway.",

author = "Licchesi, \{Julien D. F.\} and Westra, \{William H.\} and Hooker, \{Craig M.\} and Machida, \{Emi O.\} and Baylin, \{Stephen B.\} and Herman, \{James G.\}",

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T1 - Epigenetic alteration of Wnt pathway antagonists in progressive glandular neoplasia of the lung

AU - Licchesi, Julien D. F.

AU - Westra, William H.

AU - Hooker, Craig M.

AU - Machida, Emi O.

AU - Baylin, Stephen B.

AU - Herman, James G.

PY - 2008/5

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N2 - Atypical adenomatous hyperplasia (AAH) is now recognized as a precursor lesion from which lung adenocarcinomas arise and thus represents an ideal target for studying the early genetic and epigenetic alterations associated with lung tumorigenesis such as alterations of the Wnt pathway.

AB - Atypical adenomatous hyperplasia (AAH) is now recognized as a precursor lesion from which lung adenocarcinomas arise and thus represents an ideal target for studying the early genetic and epigenetic alterations associated with lung tumorigenesis such as alterations of the Wnt pathway.

UR - https://www.scopus.com/pages/publications/47249111724

UR - http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3312609/

U2 - 10.1093/carcin/bgn017

DO - 10.1093/carcin/bgn017

M3 - Article

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SN - 0143-3334

VL - 29

SP - 895

EP - 904

JO - Carcinogenesis

JF - Carcinogenesis

IS - 5

ER -

Epigenetic alteration of Wnt pathway antagonists in progressive glandular neoplasia of the lung

Abstract

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