Abstract
Atypical adenomatous hyperplasia (AAH) is now recognized as a precursor lesion from which lung adenocarcinomas arise and thus represents an ideal target for studying the early genetic and epigenetic alterations associated with lung tumorigenesis such as alterations of the Wnt pathway.
Original language | English |
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Pages (from-to) | 895-904 |
Number of pages | 10 |
Journal | Carcinogenesis |
Volume | 29 |
Issue number | 5 |
Early online date | 28 Feb 2008 |
DOIs | |
Publication status | Published - May 2008 |