Abstract
Prader-Willi syndrome (PWS) is caused by loss of paternally expressed genes at an imprinted locus on chromosome 15, including the long noncoding RNA IPW. A new study identifies a critical role for IPW in modulating the expression of maternally expressed genes in trans, which has important implications for the understanding of imprinted gene networks.
| Original language | English |
|---|---|
| Pages (from-to) | 528-530 |
| Number of pages | 3 |
| Journal | Nature Genetics |
| Volume | 46 |
| Issue number | 6 |
| Early online date | 28 May 2014 |
| DOIs | |
| Publication status | Published - Jun 2014 |
Fingerprint
Dive into the research topics of 'Cross-talk between imprinted loci in Prader-Willi syndrome'. Together they form a unique fingerprint.Cite this
- APA
- Standard
- Harvard
- Vancouver
- Author
- BIBTEX
- RIS