Cross-talk between imprinted loci in Prader-Willi syndrome

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Prader-Willi syndrome (PWS) is caused by loss of paternally expressed genes at an imprinted locus on chromosome 15, including the long noncoding RNA IPW. A new study identifies a critical role for IPW in modulating the expression of maternally expressed genes in trans, which has important implications for the understanding of imprinted gene networks.

Original languageEnglish
Pages (from-to)528-530
Number of pages3
JournalNature Genetics
Issue number6
Early online date28 May 2014
Publication statusPublished - Jun 2014


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